1
40
6
-
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
March 2024 List
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
Citation List Month
March List 2024
URL Address
<a href="http://doi.org/10.1136/bmjopen-2023-075740" target="_blank" rel="noreferrer noopener"> http://doi.org/10.1136/bmjopen-2023-075740</a>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Shepherding parents to prepare for end-of-life decision-making: a critical phenomenological study of the communication approach of paediatricians caring for children with life-limiting conditions in Australia
Publisher
An entity responsible for making the resource available
BMJ Open
Date
A point or period of time associated with an event in the lifecycle of the resource
2023
Subject
The topic of the resource
Australia; care behavior; decision making; interpersonal communication; parent; pediatrician; phenomenology; article; child; courage; human; interview; male; palliative therapy; shared decision making; signal transduction; simulation
Creator
An entity primarily responsible for making the resource
Vemuri S; Hynson J; Williams K; O'Neill J; Gillam L
Description
An account of the resource
Background/objectives: Shared decision-making is widely accepted as the best approach for end-of-life decision-making for children with life-limiting conditions. Both paediatricians and parents find benefit in preparing for such decisions. However, little detail is known about this preparatory process. This study aims to explore how paediatricians prepare parents for end-of-life decision-making for a child with a life-limiting condition using clinical simulation. Design: Individual, semistructured, post-simulation qualitative interviews of paediatricians and parent-actors. Setting: Acute intensive and long-term outpatient paediatric care in Victoria, Australia. Participants: 18 purposively sampled paediatricians who treat children with life-limiting conditions and the two parent-actors involved in all simulations. Paediatricians were excluded if they assisted in the study design, worked within specialist palliative care teams or did not provide clinical care outside the neonatal period. Results: Three key themes in a preparatory process (termed 'shepherding') were identified: (1) paediatricians aim to lead parents along a pathway to future end-of-life decisions, (2) paediatricians prefer to control the pace of these discussions and (3) paediatricians recognise they need to have courage to face risk with this preparation. Paediatricians use a variety of shepherding strategies to influence the pace, content and framing of discussions, which may help prepare parents to make the best end-of-life treatment decisions when the time comes. Conclusions: Shepherding is a newly identified, subtle process intended to influence parents by guiding their understanding of their child's health and potential suffering in advance of decision-making. Shepherding does not fit within current descriptions of physicians' decision-making influence. Paced reflection, thinking and provision of information are shepherding strategies preferred by paediatricians, and these appear the same regardless of whether paediatricians intend to steer parents towards particular treatment decisions or simply prepare them for the process of decision-making. Further study about the intention of this influence and parental perception of this communication is needed.
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1136/bmjopen-2023-075740" target="_blank" rel="noreferrer noopener">10.1136/bmjopen-2023-075740</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
2023
Article
Australia
Bmj Open
care behavior
Child
courage
Decision Making
Gillam L
Human
Hynson J
Interpersonal Communication
Interview
Male
March List 2024
O'Neill J
Palliative Therapy
Parent
Pediatrician
Phenomenology
shared decision making
Signal Transduction
Simulation
Vemuri S
Williams K
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
Citation List Month
Backlog
URL Address
<a href="http://doi.org/10.1053/j.gastro.2005.11.060" target="_blank" rel="noreferrer">http://doi.org/10.1053/j.gastro.2005.11.060</a>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Fundamentals of neurogastroenterology: basic science
Publisher
An entity responsible for making the resource available
Gastroenterology
Date
A point or period of time associated with an event in the lifecycle of the resource
2006
Subject
The topic of the resource
Humans; Animals; Brain/physiology; Signal Transduction; Gastrointestinal Motility; Neurons; Adenosine Triphosphate/physiology; Afferent/physiology; Corticotropin-Releasing Hormone/physiology; Enteric Nervous System/physiology; Excitatory Postsynaptic Potentials; Gastroenterology; Gastrointestinal Diseases/physiopathology; Neuroeffector Junction/physiology; Spinal Cord/physiology; Vagus Nerve/physiology
Creator
An entity primarily responsible for making the resource
Grundy D; Al-Chaer ED; Aziz Q; Collins SM; Ke M; Tache Y; Wood JD
Description
An account of the resource
The focus of neurogastroenterology in Rome II was the enteric nervous system (ENS). To avoid duplication with Rome II, only advances in ENS neurobiology after Rome II are reviewed together with stronger emphasis on interactions of the brain, spinal cord, and the gut in terms of relevance for abdominal pain and disordered gastrointestinal function. A committee with expertise in selective aspects of neurogastroenterology was invited to evaluate the literature and provide a consensus overview of the Fundamentals of Neurogastroenterology textbook as they relate to functional gastrointestinal disorders (FGIDs). This review is an abbreviated version of a fuller account that appears in the forthcoming book, Rome III. This report reviews current basic science understanding of visceral sensation and its modulation by inflammation and stress and advances in the neurophysiology of the ENS. Many of the concepts are derived from animal studies in which the physiologic mechanisms underlying visceral sensitivity and neural control of motility, secretion, and blood flow are examined. Impact of inflammation and stress in experimental models relative to FGIDs is reviewed as is human brain imaging, which provides a means for translating basic science to understanding FGID symptoms. Investigative evidence and emerging concepts implicate dysfunction in the nervous system as a significant factor underlying patient symptoms in FGIDs. Continued focus on neurogastroenterologic factors that underlie the development of symptoms will lead to mechanistic understanding that is expected to directly benefit the large contingent of patients and care-givers who deal with FGIDs.
2006
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1053/j.gastro.2005.11.060" target="_blank" rel="noreferrer">10.1053/j.gastro.2005.11.060</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Type
The nature or genre of the resource
Journal Article
2006
Adenosine Triphosphate/physiology
Afferent/physiology
Al-Chaer ED
Animals
Aziz Q
Backlog
Brain/physiology
Collins SM
Corticotropin-Releasing Hormone/physiology
Enteric Nervous System/physiology
Excitatory Postsynaptic Potentials
Gastroenterology
Gastrointestinal Diseases/physiopathology
Gastrointestinal Motility
Grundy D
Humans
Journal Article
Ke M
Neuroeffector Junction/physiology
Neurons
Signal Transduction
Spinal Cord/physiology
Tache Y
Vagus Nerve/physiology
Wood JD
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
Citation List Month
Backlog
URL Address
<a href="http://doi.org/10.1042/bse0390105" target="_blank" rel="noreferrer">http://doi.org/10.1042/bse0390105</a>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
The final step in programmed cell death: phagocytes carry apoptotic cells to the grave.
Publisher
An entity responsible for making the resource available
Essays In Biochemistry
Date
A point or period of time associated with an event in the lifecycle of the resource
2003
Subject
The topic of the resource
Animals; Models; Receptors; Signal Transduction; Molecular; Inflammation/metabolism; Apoptosis; Phagocytosis; Biological; Caenorhabditis elegans/genetics; Cell Surface/me [Metabolism]; Drosophila melanogaster/genetics; Evolution; Mice/genetics; Phagocytes/physiology
Creator
An entity primarily responsible for making the resource
deCathelineau AM; Henson PM
Description
An account of the resource
As cells undergo apoptosis, they are recognized and removed from the body by phagocytes. This oft-overlooked yet critical final step in the cell-death programme protects tissues from exposure to the toxic contents of dying cells and also serves to prevent further tissue damage by stimulating production of anti-inflammatory cytokines and chemokines. The clearance of apoptotic-cell corpses occurs throughout the lifespan of multicellular organisms and is important for normal development during embryogenesis, the maintenance of normal tissue integrity and function, and the resolution of inflammation. Many of the signal-transduction molecules implicated in the phagocytosis of apoptotic cells appear to have a high degree of evolutionary conservation, and therefore the engulfment of apoptotic cells is likely to represent one of the most primitive forms of phagocytosis. With the realization that the signals that govern apoptotic-cell removal also serve to attenuate inflammation and the immune response, as well as initiate signals for tissue repair and remodelling in response to cell death, the study of apoptotic cell clearance is a field experiencing a dynamic increase in interest and momentum.
2003
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1042/bse0390105" target="_blank" rel="noreferrer">10.1042/bse0390105</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Type
The nature or genre of the resource
Journal Article
2003
Animals
Apoptosis
Backlog
Biological
Caenorhabditis elegans/genetics
Cell Surface/me [Metabolism]
deCathelineau AM
Drosophila melanogaster/genetics
Essays In Biochemistry
Evolution
Henson PM
Inflammation/metabolism
Journal Article
Mice/genetics
Models
Molecular
Phagocytes/physiology
Phagocytosis
Receptors
Signal Transduction
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
Citation List Month
Backlog
URL Address
<a href="http://doi.org/10.1002/jlb.58.2.137" target="_blank" rel="noreferrer">http://doi.org/10.1002/jlb.58.2.137</a>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Associations between the neuroendocrine and immune systems
Publisher
An entity responsible for making the resource available
Journal Of Leukocyte Biology
Date
A point or period of time associated with an event in the lifecycle of the resource
1995
Subject
The topic of the resource
Humans; Animals; Biomarkers of Pain; Cell Communication; Cytokines/physiology; Hormones/physiology; Hypothalamo-Hypophyseal System/physiology; Immune System/physiology; Interleukins/physiology; Leukocytes/physiology; Neurosecretory Systems/physiology; Signal Transduction
Creator
An entity primarily responsible for making the resource
Weigent DA; Blalock JE
Description
An account of the resource
Organisms respond to infection with complex adaptations involving bidirectional communication between the immune and neuroendocrine systems. The idea of intercellular communication between the neuroendocrine and immune systems via common signal molecules has provided a conceptual framework for such crosstalk. The studies to date show that cells of the immune system contain receptors for neuroendocrine hormones and can also be considered a source of pituitary and hypothalamic peptides. The structure and pattern of synthesis of these peptides by leukocytes appear similar to neuroendocrine hormones, although some differences exist. Once secreted, these peptide hormones may function as endogenous regulators of the immune system as well as conveyors of information from the immune to the neuroendocrine system. The plasma hormone concentrations contributed by lymphocytes usually do not reach the levels required when the pituitary gland is the source, but because immune cells are mobile, they have the potential to locally deposit the hormone at the target site. Likewise, other studies show that cells of the neuroendocrine system contain receptors for cytokines and can also be considered a source of cytokines, particularly interleukin-1 (IL-1) and IL-6. In the pituitary IL-1 beta coexists with thyroid stimulating hormone in a subpopulation of thyrotropes, suggesting it may have a role as a pituitary paracrine factor. The cytokines, including IL-1, IL-2, IL-6, interferon-gamma, and tumor necrosis factor, exert profound effects on hypothalamic pituitary axes. It is our hypothesis that the relay of information to the neuroendocrine system represents a sensory function for the immune system wherein leukocytes recognize stimuli that are not recognizable by the central and peripheral nervous systems (i.e., bacteria, tumors, viruses, and antigens). The recognition of such noncognitive stimuli by immunocytes is then converted into information and a physiological change occurs. Future studies into the physiological role that cytokines and neuroendocrine hormones have in these systems will be of considerable interest for both immunologists and endocrinologists.
1995
Identifier
An unambiguous reference to the resource within a given context
<a href="http://doi.org/10.1002/jlb.58.2.137" target="_blank" rel="noreferrer">10.1002/jlb.58.2.137</a>
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Type
The nature or genre of the resource
Journal Article
1995
Animals
Backlog
Biomarkers of Pain
Blalock JE
Cell Communication
Cytokines/physiology
Hormones/physiology
Humans
Hypothalamo-Hypophyseal System/physiology
Immune System/physiology
Interleukins/physiology
Journal Article
Journal Of Leukocyte Biology
Leukocytes/physiology
Neurosecretory Systems/physiology
Signal Transduction
Weigent DA
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
Citation List Month
Backlog
URL Address
<a href="http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=10846153" target="_blank" rel="noreferrer">http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&dopt=Citation&list_uids=10846153</a>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Neuronal plasticity: increasing the gain in pain
Publisher
An entity responsible for making the resource available
Science
Date
A point or period of time associated with an event in the lifecycle of the resource
2000
Subject
The topic of the resource
Models; Human; Signal Transduction; Neurological; Inflammation/physiopathology; Animal; Nociceptors/physiology; Neurons; Pain/physiopathology; Neuronal Plasticity; Afferent/physiology; Peripheral Nerves/injuries; Posterior Horn Cells/physiology; Synaptic Transmission
Creator
An entity primarily responsible for making the resource
Woolf CJ; Salter MW
Description
An account of the resource
We describe those sensations that are unpleasant, intense, or distressing as painful. Pain is not homogeneous, however, and comprises three categories: physiological, inflammatory, and neuropathic pain. Multiple mechanisms contribute, each of which is subject to or an expression of neural plasticity-the capacity of neurons to change their function, chemical profile, or structure. Here, we develop a conceptual framework for the contribution of plasticity in primary sensory and dorsal horn neurons to the pathogenesis of pain, identifying distinct forms of plasticity, which we term activation, modulation, and modification, that by increasing gain, elicit pain hypersensitivity.
2000
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Type
The nature or genre of the resource
Journal Article
2000
Afferent/physiology
Animal
Backlog
Human
Inflammation/physiopathology
Journal Article
Models
Neurological
Neuronal Plasticity
Neurons
Nociceptors/physiology
Pain/physiopathology
Peripheral Nerves/injuries
Posterior Horn Cells/physiology
Salter MW
Science
Signal Transduction
Synaptic Transmission
Woolf CJ
-
Text
A resource consisting primarily of words for reading. Examples include books, letters, dissertations, poems, newspapers, articles, archives of mailing lists. Note that facsimiles or images of texts are still of the genre Text.
Citation List Month
November 2017 List
URL Address
http://ovidsp.ovid.com/ovidweb.cgi?T=JS&CSC=Y&NEWS=N&PAGE=fulltext&D=emex&AN=618183528
Notes
<p>Using Smart Source Parsing<br />(pp Date of Publication: 2017</p>
Dublin Core
The Dublin Core metadata element set is common to all Omeka records, including items, files, and collections. For more information see, http://dublincore.org/documents/dces/.
Title
A name given to the resource
Late Effects of Treatment and Palliative Care
Publisher
An entity responsible for making the resource available
Pediatric Oncology
Date
A point or period of time associated with an event in the lifecycle of the resource
2017
Subject
The topic of the resource
Central Nervous System Tumor; Palliative Therapy; 59-05-2 (methotrexate); 69-74-9 (cytarabine); 147-94-4 (cytarabine); 154-93-8 (carmustine); 7413-34-5 (methotrexate); 15475-56-6 (methotrexate); 15663-27-1 (cisplatin); 26035-31-4 (cisplatin); 96081-74-2 (cisplatin); Advance Care Planning; Appetite Disorder; Bone Density; Brain Tumor; Carmustine; Childhood Cancer Survivor; Cisplatin; Constipation; Corticosteroid; Cytarabine; Diarrhea; Dyspnea; Endocrine Disease; Fatigue; Genetic Polymorphism; Health Care Quality; Human; Incidence; Medical Decision Making; Medulloblastoma; Methotrexate; Morbidity; Mortality Rate; Nausea And Vomiting; Neuroectoderm Tumor; Neuropathic Pain; Neuropsychological Test; Pain Assessment; Patient Care; Phase 1 Clinical Trial (topic); Priority Journal; Psychosocial Disorder; Quality Of Life; Questionnaire; Radiation Injury; Respiration Depression; Seizure; Signal Transduction; Spasticity
Creator
An entity primarily responsible for making the resource
Chang E; Goldsby R; Mueller S; Banerjee A
Description
An account of the resource
Identifying late effects of treatment and integrating palliative care when appropriate are increasingly recognized as important elements of childhood tumor management. Patients with CNS tumors are at a high risk for mortality, and survivors have high morbidity rates related to the late effects of treatment. While intensified therapy has improved average 5-year survival in patients with pediatric brain tumors to 73 % (Ostrom et al. 2014) from less than 60 % in 1975-1979 (Linabery and Ross 2008), it has also increased the long-term consequences. Survivors may develop a spectrum of late effects ranging from subtle memory loss and cosmetic anomalies to severe neurological disabilities and recurrent neoplasms. While seemingly quite different, both palliative and late-effects care focus on improving quality of life for patients and need to be integrated into the overall care plan.
Identifier
An unambiguous reference to the resource within a given context
10.1007/978-3-319-30789-3_17
Rights
Information about rights held in and over the resource
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
147-94-4 (cytarabine)
154-93-8 (carmustine)
15475-56-6 (methotrexate)
15663-27-1 (cisplatin)
2017
26035-31-4 (cisplatin)
59-05-2 (methotrexate)
69-74-9 (cytarabine)
7413-34-5 (methotrexate)
96081-74-2 (cisplatin)
Advance Care Planning
Appetite Disorder
Banerjee A
Bone Density
Brain Tumor
Carmustine
Central Nervous System Tumor
Chang E
Childhood Cancer Survivor
Cisplatin
Constipation
Corticosteroid
Cytarabine
Diarrhea
Dyspnea
Endocrine Disease
Fatigue
Genetic Polymorphism
Goldsby R
Health Care Quality
Human
Incidence
Medical Decision Making
Medulloblastoma
Methotrexate
Morbidity
Mortality Rate
Mueller S
Nausea And Vomiting
Neuroectoderm Tumor
Neuropathic Pain
Neuropsychological Test
November 2017 List
Pain Assessment
Palliative Therapy
Patient Care
Pediatric Oncology
Phase 1 Clinical Trial (topic)
Priority Journal
Psychosocial Disorder
Quality Of Life
Questionnaire
Radiation Injury
Respiration Depression
Seizure
Signal Transduction
Spasticity