Neurologically impaired children and digestive problems
Child; Humans; Follow-Up Studies; Prospective Studies; Treatment Outcome; Fundoplication; Enteral Nutrition; adolescent; Preschool; infant; Q3 Literature Search; Injections; disabled children; Botulinum Toxin Type A/administration & dosage; Brain Damage; Chronic/complications/therapy; France; Gastric Acidity Determination; Gastroesophageal Reflux/complications/therapy; Gastroscopy; Intramuscular; Protein-Energy Malnutrition/etiology/therapy
2006
Podevin G; Capito C; Leclair MD; Heloury Y
Archives De Pediatrie
2006
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Journal Article
<a href="http://doi.org/10.1016/j.arcped.2006.03.041" target="_blank" rel="noreferrer">10.1016/j.arcped.2006.03.041</a>
Impact of feeding problems on nutritional intake and growth: Oxford Feeding Study II
Child; Female; Humans; Male; adolescent; Preschool; Nutritional Failure; Nutrition Assessment; Anthropometry; disabled children; Brain Damage; Child Nutrition Disorders/diagnosis; Chronic/diagnosis; Developmental Disabilities/diagnosis; Eating Disorders/diagnosis/etiology; Failure to Thrive/diagnosis/etiology
Poor nutritional status and growth failure are common in children with cerebral palsy (CP). The aim of this study was to assess, within a subgroup of a large and clearly defined population of children with disabilities, the impact of feeding difficulties on (1) the quality (micronutrient intake) and quantity (macronutrient intake) of their diet and (2) their growth. One hundred children with disabilities (40 females, 60 males; mean age 9 years, SD 2 years 5 months; range 4 years 6 months to 13 years 7 months) underwent a detailed dietetic analysis and a comprehensive anthropometric assessment. Diagnostic categories of disability were: CP (n=90); global developmental delay (n=3); Marfan syndrome (n=1); intractable epilepsy (n=2); agenesis of the corpus callosum (n=2); methyl malonic aciduria (n=1); and congenital rubella (n=1). Neurological impairment was classified according to difficulty with mobility which was graded as mild (little or no difficulty walking), moderate (difficulty walking but does not need aids or a helper), and severe (needs aids and/or a helper or cannot walk). Results confirmed the significant impact of neurological impairment in children on body growth and nutritional status becoming worse in those with a greater degree of motor impairment. The major nutritional deficit was in energy intake, with only one fifth reportedly regularly achieving over 100% estimated average requirement (EAR), whilst micronutrient intake was less markedly impaired and protein intake was normal in this group (96% above EAR). Many children with neurological impairment would benefit from individual nutritional assessment and management as part of their overall care.
2002
Sullivan P; Juszczak E; Lambert BR; Rose M; Ford-Adams ME; Johnson A
Developmental Medicine And Child Neurology
2002
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Journal Article
<a href="http://doi.org/10.1017/s0012162201002365" target="_blank" rel="noreferrer">10.1017/s0012162201002365</a>
Brain damage markers in children. Neurobiological and clinical aspects
Child; Female; Humans; Male; Prognosis; Sensitivity and Specificity; Predictive Value of Tests; Nerve Growth Factors; Preschool; Brain Damage; Biological Markers/blood; Brain Injuries/blood/diagnosis; Chronic/blood/diagnosis; Glial Fibrillary Acidic Protein/blood; Phosphopyruvate Hydratase/blood; S100 Proteins/blood
The presence in blood of proteins normally confined to the cytoplasm of brain cells is considered peripheral evidence of brain damage. Only recently have these proteins been measured in the blood of children at risk of brain damage. To show the value and limitations of measuring these proteins, we review their biology and the adult literature that has correlated the blood concentrations of these proteins with lesion size and dysfunction. Conclusion: We conclude that brain damage markers will increasingly be measured in the blood of newborns and other children at risk of brain damage.
2002
Leviton A; Dammann O
Acta Paediatrica
2002
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Journal Article
<a href="http://doi.org/10.1080/080352502753457851" target="_blank" rel="noreferrer">10.1080/080352502753457851</a>
Respiratory function in handicapped children
Child; Female; Male; Posture; adolescent; Preschool; Comparative Study; Human; Recurrence; Brain Damage; Brain Damage; Disabled Persons; Muscular Dystrophies/pp [Physiopathology]; Respiration Disorders/et [Etiology]; Tidal Volume; Vital Capacity; Anoxia/et [Etiology]; Chronic/co [Complications]; Chronic/pp [Physiopathology]; Hypercapnia/et [Etiology]; Motor Skills; Muscular Dystrophies/co [Complications]; Respiratory Tract Infections/et [Etiology]; Spinal Dysraphism/co [Complications]; Spinal Dysraphism/pp [Physiopathology]
The aim of this study was to evaluate respiratory function of severely handicapped children. Tidal volumes and respiratory rates were determined in a total of 130 children with different clinical motor abilities. Tidal volume of non-sitters (n = 39) was significantly lower than ambulators (n = 49) or sitters (n = 42) (p less than 0.01). There was no difference in respiratory rate among the three groups. Among 45 children whose vital capacity could be determined, the tidal volumes showed a significant correlation with vital capacity (r = 0.56, p less than 0.001). Among four children whose tidal volume was less than 200 ml and respiratory rate was more than 30 cpm, blood gas analysis revealed hypoxia in three of them. The tidal volumes, therefore, would be a useful guide to estimate respiratory functions. It was concluded that the respiratory function in a non-sitter with reduced tidal volume is impaired, and that preventive measures must be taken against respiratory infection.
1990
Ishida C; Fujita M; Umemoto H; Taneda M; Sanae N; Tasaki T
Brain & Development
1990
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Journal Article
<a href="http://doi.org/10.1016/s0387-7604(12)80067-2" target="_blank" rel="noreferrer">10.1016/s0387-7604(12)80067-2</a>
Studies on nutrition in severely neurologically disabled children in an institution
Child; Female; Humans; infant; Male; adolescent; Preschool; Nutritional Status; Newborn; Nervous System Diseases/complications; Brain Damage; Energy Intake; Epilepsy/complications; Mental Retardation/complications; Institutionalized; Nutrition Disorders/complications; Asphyxia Neonatorum/complications; Chronic/complications; Ferritin/blood; Growth Disorders/etiology; Hemoglobins/analysis; Selenium/blood; Vitamin D/blood; Vitamin E/blood
Severe neurological handicaps in children are frequently accompanied by growth retardation. We have studied 13 severely neurologically impaired children in an institution to see if their poor growth was related to a low intake of energy and nutrients, if this was reflected in biochemical nutritional parameters, and to modify their diet according to the results. The investigation showed low dietary intakes of energy and of several of the nutrients, with corresponding low Hb values and serum values of ferritin, selenium and vitamins E and D in some of the children. All the children were initially light for age, with catch-up growth after intervention. We conclude that severely disabled children are at high risk for under- and malnutrition, and that this may partly explain the growth retardation in the study group. To avoid the potential detrimental effects of malnutrition, it is important to aim at providing an optimal diet.
1996
Hals J; Ek J; Svalastog AG; Nilsen H
Acta Paediatrica
1996
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Journal Article
<a href="http://doi.org/10.1111/j.1651-2227.1996.tb13954.x" target="_blank" rel="noreferrer">10.1111/j.1651-2227.1996.tb13954.x</a>
Withdrawing artificial feeding from children with brain damage
Child; Humans; Withholding Treatment; Treatment Refusal; Euthanasia; Enteral Nutrition; Ethics; Medical; Preschool; Death and Euthanasia; Brain Diseases; Brain Damage; Chronic; Passive; Persistent Vegetative State
1995
Cranford R
Bmj (clinical Research Ed.)
1995
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
Journal Article
<a href="http://doi.org/10.1136/bmj.311.7003.464" target="_blank" rel="noreferrer">10.1136/bmj.311.7003.464</a>
Palliative Care After Neonatal Intensive Care: Contributions Of Leonetti Law And Remaining Challenges
Newborn Intensive Care; Palliative Therapy; Patient Right; Terminal Care; Artificial Feeding; Brain Damage; Climate; Clinical Practice; Clinical Study; Decision Making; Drug Withdrawal; Dying; Emotion; Ethics; Health Promotion; Human; Human Dignity; Hydration; Life Sustaining Treatment; Medical Decision Making; Medical Ethics; Medical Practice; Neonatology; Newborn; Palliative Therapy; Perinatal Care; Perinatal Nursing; Quality Of Life; Short Survey; Survival; Terminal Care; Treatment Withdrawal
Kuhn P; Dillenseger L; Cojean N; Escande B; Zores C; Astruc D
Archives De Pediatrie
2017
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
10.1016/j.arcped.2016.11.012
Family-oriented Palliative Care: Parents' Perspective And Experience
Family Study; Palliative Therapy; Brain Damage; Child; Chromosome Disorder; Chromosome Disorders; Complication; Controlled Study; Dyspnea; Gene Deletion; Hospice; Hospital; Human; Length Of Stay; Life Expectancy; Medical Service; Metabolic Disorder; Neuromuscular Disease; Only Child; Pain; Palliative Care; Quality Of Life; Seizure; Spasticity; Spiritual Care; Symptom
Palliative care is needed for children with neurodegenerative and progressive neuromuscular diseases, inborn genetic (e.g., chromosomal disorders, deletion syndromes) or metabolic disorders, as well as for children with early brain lesions which can decrease life-expectancy due to complications. Care for children with life-shortening diseases is a major challenge for parents and the whole family. A vast majority of families prefer to live at home with their severely sick child, and they also want to avoid admissions to hospital and stay at their familiar environment for the final period of life. To cope with all these demands, many families need a lot of support. By means of early integration of palliative perspective and care support can be adapted to the needs of the child and the family. Many children are living at home with their parents for many years. In many cases, quality of life and a stable familiar environment can be assured only by substantial external assistance. Support by specialized palliative care teams (SAPV), hospices and hospice services is able to unburden families. Important tasks are continuous and 24-hour medical services for symptom control (regarding e.g., pain, dyspnea, seizures, spasticity) as well as psychological, emotional, social and spiritual care for patients and all other family members.
Pietz J
Neuropediatrics
2016
Article information provided for research and reference use only. PedPalASCNET does not hold any rights over the resource listed here. All rights are retained by the journal listed under publisher and/or the creator(s).
DOI: 10.1055/s-0036-1583746